The observation of temporal hypoperfusion in adults and children with CFS / CFIDS, may define autism as a disorder of impaired relations with the surrounding environment determined by the temporal hypofunction leading as a consequence to a diaschetic hypofunction of visual cortex and cerebellum. The mechanisms for this abnormality need to be investigated using activation techniques and other approaches i.e. evaluation of possible immune dysregulation, etc.
With the general finding of a physiologic hypoperfusion / hypofunction on NeuroSPECT, with generally normal MRI’s and CAT scans, we are optimistically looking at areas of dysfunction amenable to therapy and improved return of function. Work is beginning (clinical research in progress) to define reasons for this function and in turn potential avenues of therapeutic intervention.